The question of how pain begins must involve generation of biochemical mediators that interface with the nerve terminals of nociceptive sensory neurons. A corollary of this hypothesis is that the necessary molecular apparatus for generating such algogenic mediators is present or can be induced in tissue in response to damage or pathological conditions. Framed by the view that tissue damage involves membrane damage, the present investigation examines these questions from the perspective of bioactive lipids.
from The Journal of Pain https://ift.tt/2SulyXh
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